Cargando…
B cell activation and the establishment of Epstein-Barr virus latency
Linear EBV genomes undergo a transition to the circular form characteristic of latency by 16-20 h post-infection. This transition requires that the infected cells be activated to the G1 stage of the cell cycle. Cellular proliferation and expression of the activation marker CD23 were not required. Ne...
Formato: | Texto |
---|---|
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
1988
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2189139/ https://www.ncbi.nlm.nih.gov/pubmed/2848918 |
Ejemplares similares
-
Molecular Basis of Epstein–Barr Virus Latency Establishment and Lytic Reactivation
por: Murata, Takayuki, et al.
Publicado: (2021) -
To Be or Not IIb: A Multi-Step Process for Epstein-Barr Virus Latency Establishment and Consequences for B Cell Tumorigenesis
por: Price, Alexander M., et al.
Publicado: (2015) -
Epstein–Barr virus latency programs dynamically sensitize B cells to ferroptosis
por: Burton, Eric M., et al.
Publicado: (2022) -
Identification of Host Biomarkers of Epstein-Barr Virus Latency IIb and Latency III
por: Messinger, Joshua E., et al.
Publicado: (2019) -
Direct Evidence of Abortive Lytic Infection-Mediated Establishment of Epstein-Barr Virus Latency During B-Cell Infection
por: Inagaki, Tomoki, et al.
Publicado: (2021)