Cargando…
Prevention of nephritis in major histocompatibility complex class II- deficient MRL-lpr mice
MRL-lpr mice develop aggressive autoimmune kidney disease associated with increased or de novo renal expression of major histocompatibility complex (MHC) class II molecules and a massive systemic expansion of CD4-CD- double negative (DN) T cells. Whereas non-MHC linked genes can have a profound effe...
Formato: | Texto |
---|---|
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
1994
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2191453/ https://www.ncbi.nlm.nih.gov/pubmed/7908320 |
Ejemplares similares
-
ALW peptide ameliorates lupus nephritis in MRL/lpr mice
por: Wang, Huixia, et al.
Publicado: (2019) -
Prevention of Immune Nephritis by the Small Molecular Weight Immunomodulator Iguratimod in MRL/lpr Mice
por: Yan, Qingran, et al.
Publicado: (2014) -
Reduction of lupus nephritis in MRL/lpr mice by a bacterial superantigen treatment
Publicado: (1991) -
Greatly reduced lymphoproliferation in lpr mice lacking major histocompatibility complex class I
Publicado: (1995) -
Nitric oxide as an inflammatory mediator in autoimmune MRL-lpr/lpr mice.
por: Weinberg, J B
Publicado: (1998)