Cargando…
Oligodendrocyte-specific expression and autoantigenicity of transaldolase in multiple sclerosis
Although the etiology of multiple sclerosis (MS) is unknown, there is compelling evidence that its pathogenesis is mediated through the immune system. Molecular mimicry, i.e., crossreactivity between self- antigens and viral proteins, has been implicated in the initiation of autoimmunity and MS. Bas...
Formato: | Texto |
---|---|
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
1994
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2191732/ https://www.ncbi.nlm.nih.gov/pubmed/7964452 |
Ejemplares similares
-
Transaldolase haploinsufficiency in subjects with acetaminophen‐induced liver failure
por: Oaks, Zachary, et al.
Publicado: (2020) -
Clinical and molecular characteristics of two transaldolase-deficient patients
por: Tylki-Szymanska, Anna, et al.
Publicado: (2014) -
Disease-specific oligodendrocyte lineage cells arise in multiple sclerosis
por: Falcão, Ana Mendanha, et al.
Publicado: (2019) -
Constitutive homologous expression of phosphoglucomutase and transaldolase increases the metabolic flux of Fusarium oxysporum
por: Anasontzis, George E, et al.
Publicado: (2014) -
TMEM10 Promotes Oligodendrocyte Differentiation and is Expressed by Oligodendrocytes in Human Remyelinating Multiple Sclerosis Plaques
por: de Faria, Omar, et al.
Publicado: (2019)