Cargando…
Rat blood neutrophils express very late antigen 4 and it mediates migration to arthritic joint and dermal inflammation
Blood neutrophils contribute to joint injury in human and experimental models of arthritis. Neutrophil migration out of the blood in joint inflammation involves both the CD18 (beta2) integrins and a CD18 integrin-independent pathway. To investigate this migration, radiolabeled rat blood neutrophils...
Formato: | Texto |
---|---|
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
1996
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2192571/ https://www.ncbi.nlm.nih.gov/pubmed/8642327 |
Ejemplares similares
-
PUMA gene delivery to synoviocytes reduces inflammation and degeneration of arthritic joints
por: Hong, Saw-See, et al.
Publicado: (2017) -
Conserved human effector Treg cell transcriptomic and epigenetic signature in arthritic joint inflammation
por: Mijnheer, Gerdien, et al.
Publicado: (2021) -
Methyl Jasmonate Reduces Inflammation and Oxidative Stress in the Brain of Arthritic Rats
por: Pereira-Maróstica, Heloisa V., et al.
Publicado: (2019) -
The heat-stable antigen can alter very late antigen 4-mediated adhesion
Publicado: (1994) -
Protective effect of curcumin on experimentally induced arthritic rats: detailed histopathological study of the joints and white blood cell count
por: Kamarudin, Taty Anna, et al.
Publicado: (2012)