Cargando…
B cells lacking RelB are defective in proliferative responses, but undergo normal B cell maturation to Ig secretion and Ig class switching
A number of distinct functional abnormalities have been observed in B cells derived from p50/ NF-kappa B or c-rel knockout mice. RelB, another member of the NF-kappa B/Rel family of transcription factors, is expressed during the latter stages of B cell maturation and can bind to regulatory sites wit...
Formato: | Texto |
---|---|
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
1996
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2192806/ https://www.ncbi.nlm.nih.gov/pubmed/8879226 |
Ejemplares similares
-
RelB and Neuroinflammation
por: Mockenhaupt, Karli, et al.
Publicado: (2021) -
The mitochondrial localization of RelB and NFATx in immature T cells
por: Stasik, Izabela, et al.
Publicado: (2008) -
RelB-Dependent Stromal Cells Promote T-Cell Leukemogenesis
por: dos Santos, Nuno R., et al.
Publicado: (2008) -
RelB and RelE of Escherichia coli Form a Tight Complex That Represses Transcription via the Ribbon–Helix–Helix Motif in RelB
por: Overgaard, Martin, et al.
Publicado: (2009) -
NF-κB/Rel Transcription Factors in Pancreatic Cancer: Focusing on RelA, c-Rel, and RelB
por: Kabacaoglu, Derya, et al.
Publicado: (2019)