Cargando…
Depletion of Cd4(+) T Cells Causes Reactivation of Murine Persistent Tuberculosis despite Continued Expression of Interferon γ and Nitric Oxide Synthase 2
Tuberculosis is a major cause of death in much of the world. Current estimates are that one-third of the world's population is infected with Mycobacterium tuberculosis. Most infected persons control the infection but in many cases may not eliminate the organism. Reactivation of this clinically...
Autores principales: | Scanga, Charles A., Mohan, V.P., Yu, Keming, Joseph, Heather, Tanaka, Kathryn, Chan, John, Flynn, JoAnne L. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
2000
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2193220/ https://www.ncbi.nlm.nih.gov/pubmed/10934223 |
Ejemplares similares
-
Reprogramming of the Macrophage Transcriptome in Response to Interferon-γ and Mycobacterium tuberculosis
: Signaling Roles of Nitric Oxide Synthase-2 and Phagocyte Oxidase
por: Ehrt, Sabine, et al.
Publicado: (2001) -
Role of interferon regulatory factor 1 in induction of nitric oxide synthase
Publicado: (1994) -
Characterization of Calmodulin-Free Murine Inducible Nitric-Oxide Synthase
por: Nagpal, Latika, et al.
Publicado: (2015) -
Type I Interferon Receptor Deficiency in Dendritic Cells Facilitates Systemic Murine Norovirus Persistence Despite Enhanced Adaptive Immunity
por: Nice, Timothy J., et al.
Publicado: (2016) -
Interferon-γ augments GABA release in the developing neocortex via nitric oxide synthase/soluble guanylate cyclase and constrains network activity
por: Döhne, Noah, et al.
Publicado: (2022)