Cargando…
Cc Chemokine Receptor 2 Is Critical for Induction of Experimental Autoimmune Encephalomyelitis
Experimental autoimmune encephalomyelitis (EAE) is a CD4(+) T lymphocyte–mediated disease of the central nervous system (CNS) characterized by mononuclear cell infiltration, demyelination, and paralysis. We previously demonstrated a role for chemokines in acute and relapsing EAE pathogenesis. Presen...
Autores principales: | Fife, Brian T., Huffnagle, Gary B., Kuziel, William A., Karpus, William J. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
The Rockefeller University Press
2000
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2193286/ https://www.ncbi.nlm.nih.gov/pubmed/10993920 |
Ejemplares similares
-
Resistance to Experimental Autoimmune Encephalomyelitis in Mice Lacking the Cc Chemokine Receptor (Ccr2)
por: Izikson, Leonid, et al.
Publicado: (2000) -
Transgenic Interleukin 10 Prevents Induction of Experimental Autoimmune Encephalomyelitis
por: Cua, Daniel J., et al.
Publicado: (1999) -
Tumor Necrosis Factor α and Lymphotoxin α Are Not Required for Induction of Acute Experimental Autoimmune Encephalomyelitis
por: Frei, Karl, et al.
Publicado: (1997) -
Treatment of Experimental Autoimmune Encephalomyelitis with Genetically Modified Memory T Cells
por: Mathisen, Peter M., et al.
Publicado: (1997) -
Identification of CCR8: A Human Monocyte and Thymus Receptor for the CC Chemokine I-309
por: Tiffany, H. Lee, et al.
Publicado: (1997)