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Augmentation of Recovery from Inactivation by Site-3 Na Channel Toxins : A Single-Channel and Whole-Cell Study of Persistent Currents

Site-3 toxins isolated from several species of scorpion and sea anemone bind to voltage-gated Na channels and prolong the time course of I(Na) by interfering with inactivation with little or no effect on activation, effects that have similarities to those produced by genetic diseases in skeletal mus...

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Detalles Bibliográficos
Autores principales:	Richard Benzinger, G., Tonkovich, Gayle S., Hanck, Dorothy A.
Formato:	Texto
Lenguaje:	English
Publicado:	The Rockefeller University Press 1999
Materias:	Article
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2223375/ https://www.ncbi.nlm.nih.gov/pubmed/9925828

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