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Nitric oxide, leukocytes and microvascular permeability: causality or bystanders?
Increased microvascular permeability resulting in tissue edema is a hallmark of sepsis-related microcirculatory failure, and leukocyte–endothelium interaction is thought to assume major importance in this context. However, the role of nitric oxide (NO) in the interplay of inflammation, leukocyte–end...
| Autores principales: | , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2008
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2374610/ https://www.ncbi.nlm.nih.gov/pubmed/18226179 http://dx.doi.org/10.1186/cc6214 |