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Expression of ATF3 and axonal outgrowth are impaired after delayed nerve repair
BACKGROUND: A delay in surgical nerve repair results in impaired nerve function in humans, but mechanisms behind the weakened nerve regeneration are not known. Activating transcription factor 3 (ATF3) increases the intrinsic growth state of injured neurons early after injury, but the role of long-te...
| Autores principales: | , |
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| Formato: | Texto |
| Lenguaje: | English |
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BioMed Central
2008
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| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2556676/ https://www.ncbi.nlm.nih.gov/pubmed/18801180 http://dx.doi.org/10.1186/1471-2202-9-88 |