Cargando…
Co-regulation analysis of closely linked genes identifies a highly recurrent gain on chromosome 17q25.3 in prostate cancer
BACKGROUND: Transcriptional profiling of prostate cancer (PC) has unveiled new markers of neoplasia and allowed insights into mechanisms underlying this disease. Genomewide analyses have also identified new chromosomal abnormalities associated with PC. The combination of both classes of data for the...
Autores principales: | Bermudo, Raquel, Abia, David, Ferrer, Berta, Nayach, Iracema, Benguria, Alberto, Zaballos, Ángel, del Rey, Javier, Miró, Rosa, Campo, Elías, Martínez-A, Carlos, Ortiz, Ángel R, Fernández, Pedro L, Thomson, Timothy M |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2008
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2585097/ https://www.ncbi.nlm.nih.gov/pubmed/18973659 http://dx.doi.org/10.1186/1471-2407-8-315 |
Ejemplares similares
-
6q25.1-q25.3 Microdeletion in a Chinese Girl
por: Zhong, Mian-Ling, et al.
Publicado: (2021) -
De novo deletions and duplications of 17q25.3 cause susceptibility to cardiovascular malformations
por: Probst, F. J., et al.
Publicado: (2015) -
17q25.3 copy number changes: association with neurodevelopmental disorders and cardiac malformation
por: Sahajpal, Nikhil Shri, et al.
Publicado: (2023) -
Congenital hydrocephalus and hemivertebrae associated with de novo partial monosomy 6q (6q25.3→qter)
por: Li, Y, et al.
Publicado: (2015) -
PS08. Phenotypic analysis of a mouse model for 15q25.2–25.3 deletion syndrome
por: Nomura, Jun, et al.
Publicado: (2016)