Cargando…
Dynamic, Morphotype-Specific Candida albicans β-Glucan Exposure during Infection and Drug Treatment
Candida albicans, a clinically important dimorphic fungal pathogen that can evade immune attack by masking its cell wall β-glucan from immune recognition, mutes protective host responses mediated by the Dectin-1 β-glucan receptor on innate immune cells. Although the ability of C. albicans to switch...
Autores principales: | Wheeler, Robert T., Kombe, Diana, Agarwala, Sudeep D., Fink, Gerald R. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2008
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2587227/ https://www.ncbi.nlm.nih.gov/pubmed/19057660 http://dx.doi.org/10.1371/journal.ppat.1000227 |
Ejemplares similares
-
Candida albicans β-Glucan Differentiates Human Monocytes Into a Specific Subset of Macrophages
por: Leonhardt, Julia, et al.
Publicado: (2018) -
Exposure of Candida albicans β (1,3)-glucan is promoted by activation of the Cek1 pathway
por: Chen, Tian, et al.
Publicado: (2019) -
A computational model for regulation of nanoscale glucan exposure in Candida albicans
por: Wester, Michael J., et al.
Publicado: (2017) -
Caspofungin-induced β(1,3)-glucan exposure in Candida albicans is driven by increased chitin levels
por: Wagner, Andrew S., et al.
Publicado: (2023) -
Novel Insight into Neutrophil Immune Responses by Dry Mass Determination of Candida albicans Morphotypes
por: Hosseinzadeh, Ava, et al.
Publicado: (2013)