Cargando…
Initiation Site of Ca(2+) Entry Evoked by Endoplasmic Reticulum Ca(2+) Depletion in Mouse Parotid and Pancreatic Acinar Cells
PURPOSE: In non-excitable cells, which include parotid and pancreatic acinar cells, Ca(2+) entry is triggered via a mechanism known as capacitative Ca(2+) entry, or store-operated Ca(2+) entry. This process is initiated by the perception of the filling state of endoplasmic reticulum (ER) and the dep...
Autores principales: | Jo, Hae, Byun, Hae Mi, Lee, Syng-Ill, Shin, Dong Min |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Yonsei University College of Medicine
2007
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2628100/ https://www.ncbi.nlm.nih.gov/pubmed/17594163 http://dx.doi.org/10.3349/ymj.2007.48.3.526 |
Ejemplares similares
-
Expression of Ca(2+)-dependent Synaptotagmin Isoforms in Mouse and Rat Parotid Acinar Cells
por: Jo, Hae, et al.
Publicado: (2006) -
Alteration of Expression of Ca(2+) Signaling Proteins and Adaptation of Ca(2+) Signaling in SERCA2(+/-) Mouse Parotid Acini
por: Choi, Jong-Hoon, et al.
Publicado: (2008) -
Role of Regulators of G-Protein Signaling 4 in Ca(2+) Signaling in Mouse Pancreatic Acinar Cells
por: Park, Soonhong, et al.
Publicado: (2011) -
Hydrogen peroxide inhibits Ca(2+) efflux through plasma membrane Ca(2+)-ATPase in mouse parotid acinar cells
por: Kim, Min Jae, et al.
Publicado: (2018) -
Apical Ca(2+)-activated potassium channels in mouse parotid acinar cells
por: Almassy, Janos, et al.
Publicado: (2012)