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Mouse models for preeclampsia: disruption of redox-regulated signaling

The concept that oxidative stress contributes to the development of human preeclampsia has never been tested in genetically-defined animal models. Homozygous deletion of catechol-O-methyl transferase (Comt-/-) in pregnant mice leads to human preeclampsia-like symptoms (high blood pressure, albuminur...

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Detalles Bibliográficos
Autores principales:	Banerjee, Subhasis, Randeva, Harpal, Chambers, Anne E
Formato:	Texto
Lenguaje:	English
Publicado:	BioMed Central 2009
Materias:	Hypothesis
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2632643/ https://www.ncbi.nlm.nih.gov/pubmed/19146669 http://dx.doi.org/10.1186/1477-7827-7-4

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