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Correction: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms
| Autores principales: | , , , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
Public Library of Science
2008
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2655220/ http://dx.doi.org/10.1371/annotation/682082fc-c500-414b-9f57-f9420c00c57e |
| _version_ | 1782165439398281216 |
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| author | Meidute Abaraviciene, Sandra Lundquist, Ingmar Galvanovskis, Juris Flodgren, Erik Olde, Björn Salehi, Albert |
| author_facet | Meidute Abaraviciene, Sandra Lundquist, Ingmar Galvanovskis, Juris Flodgren, Erik Olde, Björn Salehi, Albert |
| author_sort | Meidute Abaraviciene, Sandra |
| collection | PubMed |
| description | |
| format | Text |
| id | pubmed-2655220 |
| institution | National Center for Biotechnology Information |
| language | English |
| publishDate | 2008 |
| publisher | Public Library of Science |
| record_format | MEDLINE/PubMed |
| spelling | pubmed-26552202009-03-17 Correction: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms Meidute Abaraviciene, Sandra Lundquist, Ingmar Galvanovskis, Juris Flodgren, Erik Olde, Björn Salehi, Albert PLoS One Correction Public Library of Science 2008-05-20 /pmc/articles/PMC2655220/ http://dx.doi.org/10.1371/annotation/682082fc-c500-414b-9f57-f9420c00c57e Text en Public Library of Science. http://creativecommons.org/licenses/by/4.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are properly credited. |
| spellingShingle | Correction Meidute Abaraviciene, Sandra Lundquist, Ingmar Galvanovskis, Juris Flodgren, Erik Olde, Björn Salehi, Albert Correction: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms |
| title | Correction: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms |
| title_full | Correction: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms |
| title_fullStr | Correction: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms |
| title_full_unstemmed | Correction: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms |
| title_short | Correction: Palmitate-Induced β-Cell Dysfunction Is Associated with Excessive NO Production and Is Reversed by Thiazolidinedione-Mediated Inhibition of GPR40 Transduction Mechanisms |
| title_sort | correction: palmitate-induced β-cell dysfunction is associated with excessive no production and is reversed by thiazolidinedione-mediated inhibition of gpr40 transduction mechanisms |
| topic | Correction |
| url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2655220/ http://dx.doi.org/10.1371/annotation/682082fc-c500-414b-9f57-f9420c00c57e |
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