Cargando…
Increased Expression of Heat Shock Protein 72 Protects Renal Proximal Tubular Cells from Gentamicin-induced Injury
The nephrotoxicity of gentamicin (GM) has been widely recognized. Heat shock protein 72 (HSP72) has been reported to be a cytoprotectant. However, its cytoprotective effect against GM induced kidney injury has not yet been studied. In this study, we investigated the cytoprotective effect of HSP72 on...
Autores principales: | Wang, Zhipeng, Liu, Li, Mei, Qibing, Liu, Linna, Ran, Yuhua, Zhang, Rong |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
The Korean Academy of Medical Sciences
2006
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2722003/ https://www.ncbi.nlm.nih.gov/pubmed/17043427 http://dx.doi.org/10.3346/jkms.2006.21.5.904 |
Ejemplares similares
-
Gentamicin Induced Apoptosis of Renal Tubular Epithelial (LLC-PK(1)) Cells
por: Choi, Kyu Hun, et al.
Publicado: (2000) -
Edible Bird Nest Protects the Kidney From Gentamicin Induced Acute Tubular Necrosis
por: Lim, Christopher T S., et al.
Publicado: (2021) -
Protective role of renal proximal tubular alpha-synuclein in the pathogenesis of kidney fibrosis
por: Bozic, Milica, et al.
Publicado: (2020) -
Protection of renal tubules against gentamicin induced nephrotoxicity
por: Tavafi, Majid
Publicado: (2013) -
Heat shock protein 72 suppresses apoptosis by increasing the stability of X-linked inhibitor of apoptosis protein in renal ischemia/reperfusion injury
por: ZHANG, BAIYU, et al.
Publicado: (2015)