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NNZ-2566 treatment inhibits neuroinflammation and pro-inflammatory cytokine expression induced by experimental penetrating ballistic-like brain injury in rats

BACKGROUND: Inflammatory cytokines play a crucial role in the pathophysiology of traumatic brain injury (TBI), exerting either deleterious effects on the progression of tissue damage or beneficial roles during recovery and repair. NNZ-2566, a synthetic analogue of the neuroprotective tripeptide Glyp...

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Detalles Bibliográficos
Autores principales: Wei, Hans H, Lu, Xi-Chun M, Shear, Deborah A, Waghray, Anu, Yao, Changping, Tortella, Frank C, Dave, Jitendra R
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2009
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2731085/
https://www.ncbi.nlm.nih.gov/pubmed/19656406
http://dx.doi.org/10.1186/1742-2094-6-19