CD14 Signaling Restrains Chronic Inflammation through Induction of p38-MAPK/SOCS-Dependent Tolerance

Current thinking emphasizes the primacy of CD14 in facilitating recognition of microbes by certain TLRs to initiate pro-inflammatory signaling events and the importance of p38-MAPK in augmenting such responses. Herein, this paradigm is challenged by demonstrating that recognition of live Borrelia bu...

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Detalles Bibliográficos
Autores principales: Sahay, Bikash, Patsey, Rebeca L., Eggers, Christian H., Salazar, Juan C., Radolf, Justin D., Sellati, Timothy J.
Formato: Texto
Lenguaje:English
Publicado: Public Library of Science 2009
Materias:
Research Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2781632/
https://www.ncbi.nlm.nih.gov/pubmed/20011115
http://dx.doi.org/10.1371/journal.ppat.1000687

Internet

https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2781632/
https://www.ncbi.nlm.nih.gov/pubmed/20011115
http://dx.doi.org/10.1371/journal.ppat.1000687

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