Cargando…
Ciprofloxacin Causes Persister Formation by Inducing the TisB toxin in Escherichia coli
Bacteria induce stress responses that protect the cell from lethal factors such as DNA-damaging agents. Bacterial populations also form persisters, dormant cells that are highly tolerant to antibiotics and play an important role in recalcitrance of biofilm infections. Stress response and dormancy ap...
Autores principales: | Dörr, Tobias, Vulić, Marin, Lewis, Kim |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2010
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2826370/ https://www.ncbi.nlm.nih.gov/pubmed/20186264 http://dx.doi.org/10.1371/journal.pbio.1000317 |
Ejemplares similares
-
Elevated Expression of Toxin TisB Protects Persister Cells against Ciprofloxacin but Enhances Susceptibility to Mitomycin C
por: Edelmann, Daniel, et al.
Publicado: (2021) -
An RNA pseudoknot is essential for standby-mediated translation of the tisB toxin mRNA in Escherichia coli
por: Romilly, Cédric, et al.
Publicado: (2020) -
A Shift in Perspective: A Role for the Type I Toxin TisB as Persistence-Stabilizing Factor
por: Edelmann, Daniel, et al.
Publicado: (2022) -
TisB Protein Protects Escherichia coli Cells Suffering Massive DNA Damage from Environmental Toxic Compounds
por: Su, Wei-Lin, et al.
Publicado: (2022) -
SOS Response Induces Persistence to Fluoroquinolones in Escherichia coli
por: Dörr, Tobias, et al.
Publicado: (2009)