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Normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report

The concept of compression of the rostral ventrolateral medulla as a cause for hypertension is gaining more and more interest. This report is about a 36-year-old male with a three years history of hypertension who presented with a posterior fossa mass suggestive of a hemangioblastoma. Laboratory and...

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Detalles Bibliográficos
Autores principales: Saberi, Hooshang, Meybodi, Ali Tayebi, Zeinalizadeh, Mehdi
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2009
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2827080/
https://www.ncbi.nlm.nih.gov/pubmed/20181189
http://dx.doi.org/10.1186/1757-1626-2-7106
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author Saberi, Hooshang
Meybodi, Ali Tayebi
Zeinalizadeh, Mehdi
author_facet Saberi, Hooshang
Meybodi, Ali Tayebi
Zeinalizadeh, Mehdi
author_sort Saberi, Hooshang
collection PubMed
description The concept of compression of the rostral ventrolateral medulla as a cause for hypertension is gaining more and more interest. This report is about a 36-year-old male with a three years history of hypertension who presented with a posterior fossa mass suggestive of a hemangioblastoma. Laboratory and imaging studies ruled out the presence of von Hippel-Lindau disease and/or concomitant pheochromocytoma. Post-surgical blood pressure monitoring revealed a 40 mmHg decline in blood pressure. It could be hypothesized that alleviation the compressive effect of the tumour on the rostral ventrolateral medulla as proposed by previous studies could be a contributing factor.
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spelling pubmed-28270802010-02-24 Normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report Saberi, Hooshang Meybodi, Ali Tayebi Zeinalizadeh, Mehdi Cases J Research article The concept of compression of the rostral ventrolateral medulla as a cause for hypertension is gaining more and more interest. This report is about a 36-year-old male with a three years history of hypertension who presented with a posterior fossa mass suggestive of a hemangioblastoma. Laboratory and imaging studies ruled out the presence of von Hippel-Lindau disease and/or concomitant pheochromocytoma. Post-surgical blood pressure monitoring revealed a 40 mmHg decline in blood pressure. It could be hypothesized that alleviation the compressive effect of the tumour on the rostral ventrolateral medulla as proposed by previous studies could be a contributing factor. BioMed Central 2009-04-29 /pmc/articles/PMC2827080/ /pubmed/20181189 http://dx.doi.org/10.1186/1757-1626-2-7106 Text en Copyright ©2009 Saberi et al; licensee Cases Network Ltd. licensee BioMed Central Ltd. http://creativecommons.org/licenses/by/3.0 This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Research article
Saberi, Hooshang
Meybodi, Ali Tayebi
Zeinalizadeh, Mehdi
Normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report
title Normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report
title_full Normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report
title_fullStr Normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report
title_full_unstemmed Normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report
title_short Normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report
title_sort normalization of systemic arterial hypertension following removal of posterior fossa hemangioblastoma: a case report
topic Research article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2827080/
https://www.ncbi.nlm.nih.gov/pubmed/20181189
http://dx.doi.org/10.1186/1757-1626-2-7106
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