Cargando…
Sleep triggered by an immune response in Drosophila is regulated by the circadian clock and requires the NFκB Relish
BACKGROUND: Immune challenge impacts behavior in many species. In mammals, this adaptive behavior is often manifested as an increase in sleep. Sleep has therefore been proposed to benefit the host by enhancing immune function and thereby overcome the challenge. To facilitate genetic studies on the r...
Autores principales: | Kuo, Tzu-Hsing, Pike, Douglas H, Beizaeipour, Zahra, Williams, Julie A |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2010
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2831041/ https://www.ncbi.nlm.nih.gov/pubmed/20144235 http://dx.doi.org/10.1186/1471-2202-11-17 |
Ejemplares similares
-
Polydnavirus Ank Proteins Bind NF-κB Homodimers and Inhibit Processing of Relish
por: Bitra, Kavita, et al.
Publicado: (2012) -
The NF-κB Factor Relish Regulates Atg1
Expression and Controls Autophagy
por: Nandy, Anubhab, et al.
Publicado: (2018) -
An NF-κB–driven lncRNA orchestrates colitis and circadian clock
por: Wang, Shuai, et al.
Publicado: (2020) -
NF-κB modifies the mammalian circadian clock through interaction with the core clock protein BMAL1
por: Shen, Yang, et al.
Publicado: (2021) -
Mutant huntingtin disturbs circadian clock gene expression and sleep patterns in Drosophila
por: Faragó, Anikó, et al.
Publicado: (2019)