Cargando…
TNF dependency of IL-17-induced joint pathology can be circumvented by Toll-like receptor-2 signaling
Autores principales: | Van den Berg, W, Koenders, M, van Lent, P, Joosten, L, Lubberts, E |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2003
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2833832/ http://dx.doi.org/10.1186/ar842 |
Ejemplares similares
-
IL-1-dependent cartilage damage in a macrophage-driven arthritis model can be circumvented by T cell IL-17
por: Koenders, MI, et al.
Publicado: (2004) -
TNF-α dependency of IL-17-induced joint pathology differs under naive and arthritis conditions in vivo
por: Koenders, M, et al.
Publicado: (2003) -
Neutralizing IL-17 during re-activation of experimental arthritis prevents joint inflammation and bone erosion by decreasing RANKL and IL-1
por: Koenders, MI, et al.
Publicado: (2005) -
IL-17 produced by Paneth cells drives TNF-induced shock
por: Takahashi, Nozomi, et al.
Publicado: (2008) -
IL-17 derived from juxta-articular bone and synovium contributes to joint degradation in rheumatoid arthritis
por: Chabaud, Martine, et al.
Publicado: (2001)