PhenomiR: a knowledgebase for microRNA expression in diseases and biological processes

In recent years, microRNAs have been shown to play important roles in physiological as well as malignant processes. The PhenomiR database http://mips.helmholtz-muenchen.de/phenomir provides data from 542 studies that investigate deregulation of microRNA expression in diseases and biological processe...

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Autores principales: Ruepp, Andreas, Kowarsch, Andreas, Schmidl, Daniel, Buggenthin, Felix, Brauner, Barbara, Dunger, Irmtraud, Fobo, Gisela, Frishman, Goar, Montrone, Corinna, Theis, Fabian J
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2010
Materias:
Software
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2847718/
https://www.ncbi.nlm.nih.gov/pubmed/20089154
http://dx.doi.org/10.1186/gb-2010-11-1-r6
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author Ruepp, Andreas
Kowarsch, Andreas
Schmidl, Daniel
Buggenthin, Felix
Brauner, Barbara
Dunger, Irmtraud
Fobo, Gisela
Frishman, Goar
Montrone, Corinna
Theis, Fabian J
author_facet Ruepp, Andreas
Kowarsch, Andreas
Schmidl, Daniel
Buggenthin, Felix
Brauner, Barbara
Dunger, Irmtraud
Fobo, Gisela
Frishman, Goar
Montrone, Corinna
Theis, Fabian J
author_sort Ruepp, Andreas
collection PubMed
description In recent years, microRNAs have been shown to play important roles in physiological as well as malignant processes. The PhenomiR database http://mips.helmholtz-muenchen.de/phenomir provides data from 542 studies that investigate deregulation of microRNA expression in diseases and biological processes as a systematic, manually curated resource. Using the PhenomiR dataset, we could demonstrate that, depending on disease type, independent information from cell culture studies contrasts with conclusions drawn from patient studies.
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spelling pubmed-28477182010-03-31 PhenomiR: a knowledgebase for microRNA expression in diseases and biological processes Ruepp, Andreas Kowarsch, Andreas Schmidl, Daniel Buggenthin, Felix Brauner, Barbara Dunger, Irmtraud Fobo, Gisela Frishman, Goar Montrone, Corinna Theis, Fabian J Genome Biol Software In recent years, microRNAs have been shown to play important roles in physiological as well as malignant processes. The PhenomiR database http://mips.helmholtz-muenchen.de/phenomir provides data from 542 studies that investigate deregulation of microRNA expression in diseases and biological processes as a systematic, manually curated resource. Using the PhenomiR dataset, we could demonstrate that, depending on disease type, independent information from cell culture studies contrasts with conclusions drawn from patient studies. BioMed Central 2010 2010-01-20 /pmc/articles/PMC2847718/ /pubmed/20089154 http://dx.doi.org/10.1186/gb-2010-11-1-r6 Text en Copyright ©2010 Ruepp et al.; licensee BioMed Central Ltd. http://creativecommons.org/licenses/by/2.0 This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Software
Ruepp, Andreas
Kowarsch, Andreas
Schmidl, Daniel
Buggenthin, Felix
Brauner, Barbara
Dunger, Irmtraud
Fobo, Gisela
Frishman, Goar
Montrone, Corinna
Theis, Fabian J
PhenomiR: a knowledgebase for microRNA expression in diseases and biological processes
title PhenomiR: a knowledgebase for microRNA expression in diseases and biological processes
title_full PhenomiR: a knowledgebase for microRNA expression in diseases and biological processes
title_fullStr PhenomiR: a knowledgebase for microRNA expression in diseases and biological processes
title_full_unstemmed PhenomiR: a knowledgebase for microRNA expression in diseases and biological processes
title_short PhenomiR: a knowledgebase for microRNA expression in diseases and biological processes
title_sort phenomir: a knowledgebase for microrna expression in diseases and biological processes
topic Software
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2847718/
https://www.ncbi.nlm.nih.gov/pubmed/20089154
http://dx.doi.org/10.1186/gb-2010-11-1-r6
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