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The eSS rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver

BACKGROUND: eSS is a rat model of type 2 diabetes characterized by fasting hyperglycemia, glucose intolerance, hyperinsulinemia and early hypertriglyceridemia. Diabetic symptoms worsen during the second year of life as insulin release decreases. In 12-month-old males a diffuse hepatic steatosis was...

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Autores principales: Daniele, Stella M, Montenegro, Silvana M, Tarres, María C, Picena, Juan C, Martinez, Stella M
Formato: Texto
Lenguaje:English
Publicado: BioMed Central 2010
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2847988/
https://www.ncbi.nlm.nih.gov/pubmed/20236525
http://dx.doi.org/10.1186/1758-5996-2-15
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author Daniele, Stella M
Montenegro, Silvana M
Tarres, María C
Picena, Juan C
Martinez, Stella M
author_facet Daniele, Stella M
Montenegro, Silvana M
Tarres, María C
Picena, Juan C
Martinez, Stella M
author_sort Daniele, Stella M
collection PubMed
description BACKGROUND: eSS is a rat model of type 2 diabetes characterized by fasting hyperglycemia, glucose intolerance, hyperinsulinemia and early hypertriglyceridemia. Diabetic symptoms worsen during the second year of life as insulin release decreases. In 12-month-old males a diffuse hepatic steatosis was detected. We report the disturbances of lipid metabolism of the model with regard to the diabetic syndrome. METHODS: The study was conducted in eight 12-month-old eSS male rats and seven age/weight matched eumetabolic Wistar rats fed with a complete commercial diet al libitum. Fasting plasmatic glucose, insulin, triglycerides, total cholesterol, low-density and high-density lipoprotein, and nonesterified fatty acids levels were measured. Very low density and intermediate-density lipoproteins were analyzed and hepatic lipase activity was determined. RESULTS: eSS rats developed hyperglycemia and hyperinsulinemia, indicating insulin resistance. Compared with controls, diabetic rats exhibited high plasmatic levels of NEFA, triglycerides (TG), total cholesterol (Chol) and LDL-Chol while high-density lipoprotein (HDL) cholesterol values were reduced. eSS rats also displayed TG-rich VLDL and IDL particles without changes in hepatic lipase activity. CONCLUSION: The nonobese eSS rats develop a syndrome characterized by glucose and lipid disorders and hepatic steatosis that may provide new opportunities for studying the pathogenesis of human type 2 diabetes.
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spelling pubmed-28479882010-04-01 The eSS rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver Daniele, Stella M Montenegro, Silvana M Tarres, María C Picena, Juan C Martinez, Stella M Diabetol Metab Syndr Research BACKGROUND: eSS is a rat model of type 2 diabetes characterized by fasting hyperglycemia, glucose intolerance, hyperinsulinemia and early hypertriglyceridemia. Diabetic symptoms worsen during the second year of life as insulin release decreases. In 12-month-old males a diffuse hepatic steatosis was detected. We report the disturbances of lipid metabolism of the model with regard to the diabetic syndrome. METHODS: The study was conducted in eight 12-month-old eSS male rats and seven age/weight matched eumetabolic Wistar rats fed with a complete commercial diet al libitum. Fasting plasmatic glucose, insulin, triglycerides, total cholesterol, low-density and high-density lipoprotein, and nonesterified fatty acids levels were measured. Very low density and intermediate-density lipoproteins were analyzed and hepatic lipase activity was determined. RESULTS: eSS rats developed hyperglycemia and hyperinsulinemia, indicating insulin resistance. Compared with controls, diabetic rats exhibited high plasmatic levels of NEFA, triglycerides (TG), total cholesterol (Chol) and LDL-Chol while high-density lipoprotein (HDL) cholesterol values were reduced. eSS rats also displayed TG-rich VLDL and IDL particles without changes in hepatic lipase activity. CONCLUSION: The nonobese eSS rats develop a syndrome characterized by glucose and lipid disorders and hepatic steatosis that may provide new opportunities for studying the pathogenesis of human type 2 diabetes. BioMed Central 2010-03-17 /pmc/articles/PMC2847988/ /pubmed/20236525 http://dx.doi.org/10.1186/1758-5996-2-15 Text en Copyright ©2010 Daniele et al; licensee BioMed Central Ltd. http://creativecommons.org/licenses/by/2.0 This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Research
Daniele, Stella M
Montenegro, Silvana M
Tarres, María C
Picena, Juan C
Martinez, Stella M
The eSS rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver
title The eSS rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver
title_full The eSS rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver
title_fullStr The eSS rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver
title_full_unstemmed The eSS rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver
title_short The eSS rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver
title_sort ess rat, a nonobese model of disordered glucose and lipid metabolism and fatty liver
topic Research
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2847988/
https://www.ncbi.nlm.nih.gov/pubmed/20236525
http://dx.doi.org/10.1186/1758-5996-2-15
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