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Delayed-Onset Continuous Bruxism with Olivary Hypertrophy After Top of the Basilar Syndrome

Delayed-onset continuous bruxism due to brain stem infarction has not yet been reported. A 49-year old man presented with quadriplegia and ophthalmoplegia. Brain MRI showed acute infarction in the bilateral midbrain, right thalamus and the superior cerebellum. One month later, the patient developed...

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Autores principales: Kim, Seon-Gyung, Kim, Min-Ju, Cha, Myong-Jin, Cho, Soo-Jin, Kwon, Ki-Han, Minn, Yang-Ki
Formato: Texto
Lenguaje:English
Publicado: Korean Neurological Association 2006
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2854967/
https://www.ncbi.nlm.nih.gov/pubmed/20396509
http://dx.doi.org/10.3988/jcn.2006.2.3.206
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author Kim, Seon-Gyung
Kim, Min-Ju
Cha, Myong-Jin
Cho, Soo-Jin
Kwon, Ki-Han
Minn, Yang-Ki
author_facet Kim, Seon-Gyung
Kim, Min-Ju
Cha, Myong-Jin
Cho, Soo-Jin
Kwon, Ki-Han
Minn, Yang-Ki
author_sort Kim, Seon-Gyung
collection PubMed
description Delayed-onset continuous bruxism due to brain stem infarction has not yet been reported. A 49-year old man presented with quadriplegia and ophthalmoplegia. Brain MRI showed acute infarction in the bilateral midbrain, right thalamus and the superior cerebellum. One month later, the patient developed bruxism which persisted during sleep. A palatal myoclonus was not observed. Follow up MRI taken 4 months later showed bilateral olivary hypertrophy. We suggest that the patient's bruxism may be related to the olivary hypertrophy. The bruxism generator may be located in the pontine-reticular-formation (PRF). Bilateral large midbrain lesions interrupting the cortical inhibition may have produced bilateral olivary hypertrophy, which could stimulate the PRF, producing continuous bruxism.
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spelling pubmed-28549672010-04-15 Delayed-Onset Continuous Bruxism with Olivary Hypertrophy After Top of the Basilar Syndrome Kim, Seon-Gyung Kim, Min-Ju Cha, Myong-Jin Cho, Soo-Jin Kwon, Ki-Han Minn, Yang-Ki J Clin Neurol Case Report Delayed-onset continuous bruxism due to brain stem infarction has not yet been reported. A 49-year old man presented with quadriplegia and ophthalmoplegia. Brain MRI showed acute infarction in the bilateral midbrain, right thalamus and the superior cerebellum. One month later, the patient developed bruxism which persisted during sleep. A palatal myoclonus was not observed. Follow up MRI taken 4 months later showed bilateral olivary hypertrophy. We suggest that the patient's bruxism may be related to the olivary hypertrophy. The bruxism generator may be located in the pontine-reticular-formation (PRF). Bilateral large midbrain lesions interrupting the cortical inhibition may have produced bilateral olivary hypertrophy, which could stimulate the PRF, producing continuous bruxism. Korean Neurological Association 2006-09 2006-09-20 /pmc/articles/PMC2854967/ /pubmed/20396509 http://dx.doi.org/10.3988/jcn.2006.2.3.206 Text en Copyright © 2006 Korean Neurological Association
spellingShingle Case Report
Kim, Seon-Gyung
Kim, Min-Ju
Cha, Myong-Jin
Cho, Soo-Jin
Kwon, Ki-Han
Minn, Yang-Ki
Delayed-Onset Continuous Bruxism with Olivary Hypertrophy After Top of the Basilar Syndrome
title Delayed-Onset Continuous Bruxism with Olivary Hypertrophy After Top of the Basilar Syndrome
title_full Delayed-Onset Continuous Bruxism with Olivary Hypertrophy After Top of the Basilar Syndrome
title_fullStr Delayed-Onset Continuous Bruxism with Olivary Hypertrophy After Top of the Basilar Syndrome
title_full_unstemmed Delayed-Onset Continuous Bruxism with Olivary Hypertrophy After Top of the Basilar Syndrome
title_short Delayed-Onset Continuous Bruxism with Olivary Hypertrophy After Top of the Basilar Syndrome
title_sort delayed-onset continuous bruxism with olivary hypertrophy after top of the basilar syndrome
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2854967/
https://www.ncbi.nlm.nih.gov/pubmed/20396509
http://dx.doi.org/10.3988/jcn.2006.2.3.206
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