Cargando…
Antibody limits in vivo murid herpesvirus-4 replication by IgG Fc receptor-dependent functions
Antibody is an important antiviral defence. However, it is considered to do little against human gamma-herpesviruses, which establish predominantly latent infections regulated by T cells. One limitation on analysing these infections has been that latency is already well-established at clinical prese...
Autores principales: | Wright, Debbie E., Colaco, Susanna, Colaco, Camilo, Stevenson, Philip G. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Society for General Microbiology
2009
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2885036/ https://www.ncbi.nlm.nih.gov/pubmed/19625459 http://dx.doi.org/10.1099/vir.0.014266-0 |
Ejemplares similares
-
In vivo function of the murid herpesvirus-4 ribonucleotide reductase small subunit
por: Milho, Ricardo, et al.
Publicado: (2011) -
Glycoprotein B switches conformation during murid herpesvirus 4 entry
por: Gillet, Laurent, et al.
Publicado: (2008) -
Glycoprotein L sets the neutralization profile of murid herpesvirus 4
por: Gillet, Laurent, et al.
Publicado: (2009) -
The Murid Herpesvirus-4 gH/gL Binds to Glycosaminoglycans
por: Gillet, Laurent, et al.
Publicado: (2008) -
The Murid Herpesvirus-4 gL Regulates an Entry-Associated Conformation Change in gH
por: Gillet, Laurent, et al.
Publicado: (2008)