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A novel pathway for receptor-mediated post-translational activation of inducible nitric oxide synthase
Inducible nitric oxide synthase (iNOS) is a major source of nitric oxide during inflammation whose activity is thought to be controlled primarily at the expression level. The B1 kinin receptor (B1R) post-translationally activates iNOS beyond its basal activity via extracellular signal regulated kina...
| Autores principales: | , , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
Blackwell Publishing Ltd
2011
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2888614/ https://www.ncbi.nlm.nih.gov/pubmed/20015194 http://dx.doi.org/10.1111/j.1582-4934.2009.00992.x |