Cargando…
Cross-Talk Between Mitochondria and Proteasome in Parkinson's Disease Pathogenesis
Parkinson's disease (PD) is the most common progressive neurodegenerative movement disorder, characterized by the selective loss of nigrostriatal dopaminergic neurons, and the presence of intracellular insoluble proteinaceous inclusions, known as Lewy Bodies. Although PD etiopathogenesis remain...
Autores principales: | Branco, Diogo Martins, Arduino, Daniela M., Esteves, A. Raquel, Silva, Diana F. F., Cardoso, Sandra M., Oliveira, Catarina Resende |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Frontiers Research Foundation
2010
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2890153/ https://www.ncbi.nlm.nih.gov/pubmed/20577640 http://dx.doi.org/10.3389/fnagi.2010.00017 |
Ejemplares similares
-
Mitochondrial Fusion/Fission, Transport and Autophagy in Parkinson's Disease: When Mitochondria Get Nasty
por: Arduíno, Daniela M., et al.
Publicado: (2011) -
Mitochondrial metabolism in Parkinson's disease impairs quality control autophagy by hampering microtubule-dependent traffic
por: Arduíno, Daniela M., et al.
Publicado: (2012) -
The Role of Bacteria–Mitochondria Communication in the Activation of Neuronal Innate Immunity: Implications to Parkinson’s Disease
por: Magalhães, João D., et al.
Publicado: (2023) -
Microtubule Depolymerization Potentiates Alpha-Synuclein Oligomerization
por: Esteves, A. Raquel, et al.
Publicado: (2010) -
LPS-induced mitochondrial dysfunction regulates innate immunity activation and α-synuclein oligomerization in Parkinson's disease
por: Esteves, A. Raquel, et al.
Publicado: (2023)