Cargando…
Valproate Administered after Traumatic Brain Injury Provides Neuroprotection and Improves Cognitive Function in Rats
BACKGROUND: Traumatic brain injury (TBI) initiates a complex series of neurochemical and signaling changes that lead to pathological events including neuronal hyperactivity, excessive glutamate release, inflammation, increased blood-brain barrier (BBB) permeability and cerebral edema, altered gene e...
Autores principales: | Dash, Pramod K., Orsi, Sara A., Zhang, Min, Grill, Raymond J., Pati, Shibani, Zhao, Jing, Moore, Anthony N. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2010
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2894851/ https://www.ncbi.nlm.nih.gov/pubmed/20614021 http://dx.doi.org/10.1371/journal.pone.0011383 |
Ejemplares similares
-
Involvement of the Glycogen Synthase Kinase-3 Signaling Pathway in TBI Pathology and Neurocognitive Outcome
por: Dash, Pramod K., et al.
Publicado: (2011) -
Targeting repair of the vascular endothelium and glycocalyx after traumatic injury with plasma and platelet resuscitation
por: Barry, Mark, et al.
Publicado: (2022) -
Histopathological and Biochemical Assessment of Neuroprotective Effects of Sodium Valproate and Lutein on the Pilocarpine Albino Rat Model of Epilepsy
por: Al-Rafiah, Aziza Rashed, et al.
Publicado: (2021) -
Acute administration of lithium, but not valproate, modulates cognitive judgment bias in rats
por: Rygula, Rafal, et al.
Publicado: (2014) -
2-BFI Provides Neuroprotection Against Inflammation and Necroptosis in a Rat Model of Traumatic Brain Injury
por: Ni, Haibo, et al.
Publicado: (2019)