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Synaptotoxicity of Alzheimer Beta Amyloid Can Be Explained by Its Membrane Perforating Property
The mechanisms that induce Alzheimer's disease (AD) are largely unknown thereby deterring the development of disease-modifying therapies. One working hypothesis of AD is that Aβ excess disrupts membranes causing pore formation leading to alterations in ionic homeostasis. However, it is largely...
| Autores principales: | , , , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
Public Library of Science
2010
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2910737/ https://www.ncbi.nlm.nih.gov/pubmed/20676404 http://dx.doi.org/10.1371/journal.pone.0011820 |