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Astrocytic gap junctional communication is reduced in amyloid-β-treated cultured astrocytes, but not in Alzheimer's disease transgenic mice

Alzheimer's disease is characterized by accumulation of amyloid deposits in brain, progressive cognitive deficits and reduced glucose utilization. Many consequences of the disease are attributed to neuronal dysfunction, but roles of astrocytes in its pathogenesis are not well understood. Astroc...

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Detalles Bibliográficos
Autores principales: Cruz, Nancy F, Ball, Kelly K, Dienel, Gerald A
Formato: Texto
Lenguaje:English
Publicado: American Society for Neurochemistry 2010
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2922840/
https://www.ncbi.nlm.nih.gov/pubmed/20730033
http://dx.doi.org/10.1042/AN20100017

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