Cargando…
Generation and Characterization of the Anp32e-Deficient Mouse
BACKGROUND: Accumulated literature suggests that the acidic nuclear phosphoprotein 32 kilodalton (Anp32) proteins control multiple cellular activities through different molecular mechanisms. Like other Anp32 family members, Anp32e (a.k.a. Cpd1, PhapIII) has been conserved throughout vertebrate evolu...
Autores principales: | Reilly, Patrick T., Afzal, Samia, Wakeham, Andrew, Haight, Jillian, You-Ten, Annick, Zaugg, Kathrin, Dembowy, Joanna, Young, Ashley, Mak, Tak W. |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2010
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC2964292/ https://www.ncbi.nlm.nih.gov/pubmed/21049064 http://dx.doi.org/10.1371/journal.pone.0013597 |
Ejemplares similares
-
Targeted ANP32E Mutant Mice Do Not Demonstrate Obvious Movement Defects
por: Wong, Peiyan, et al.
Publicado: (2013) -
ANP32B Deficiency Protects Mice From Lethal Influenza A Virus Challenge by Dampening the Host Immune Response
por: Beck, Sebastian, et al.
Publicado: (2020) -
ANP32B deficiency impairs proliferation and suppresses tumor progression by regulating AKT phosphorylation
por: Yang, S, et al.
Publicado: (2016) -
BALB/c-congenic ANP32B-deficient mice reveal a modifying locus that
determines viability
por: Leo, Vonny I., et al.
Publicado: (2015) -
Mammalian ANP32A and ANP32B Proteins Drive Differential Polymerase Adaptations in Avian Influenza Virus
por: Peacock, Thomas P., et al.
Publicado: (2023)