Cargando…
Intracranial hypertension: classification and patterns of evolution
Intracranial hypertension (ICH) was systematized in four categories according to its aetiology and pathogenic mechanisms: parenchymatous ICH with an intrinsic cerebral cause; vascular ICH, which has its aetiology in disorders of cerebral blood circulation; ICH caused by disorders of cerebro–spinal f...
Autores principales: | , |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Carol Davila University Press
2008
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3018963/ https://www.ncbi.nlm.nih.gov/pubmed/20108456 |
_version_ | 1782196130173419520 |
---|---|
author | Iencean, SM Ciurea, AV |
author_facet | Iencean, SM Ciurea, AV |
author_sort | Iencean, SM |
collection | PubMed |
description | Intracranial hypertension (ICH) was systematized in four categories according to its aetiology and pathogenic mechanisms: parenchymatous ICH with an intrinsic cerebral cause; vascular ICH, which has its aetiology in disorders of cerebral blood circulation; ICH caused by disorders of cerebro–spinal fluid dynamics and idiopathic ICH. The increase of intracranial pressure is the first to happen and then intracranial hypertension develops from this initial effect becoming symptomatic; it then acquires its individuality, surpassing the initial disease. The intracranial hypertension syndrome corresponds to the stage at which the increased intracranial pressure can be compensated and the acute form of intracranial hypertension is equivalent to a decompensated ICH syndrome. The decompensation of intracranial hypertension is a condition of instability and appears when the normal intrinsic ratio of intracranial pressure – time fluctuation is changed. The essential conditions for decompensation of intracranial hypertension are: the speed of intracranial pressure increase over normal values, the highest value of abnormal intracranial pressure and the duration of high ICP values. Medical objectives are preventing ICP from exceeding 20 mm Hg and maintaining a normal cerebral blood flow. The emergency therapy is the same for the acute form but each of the four forms of ICH has a specific therapy, according to the pathogenic mechanism and if possible to aetiology. |
format | Text |
id | pubmed-3018963 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2008 |
publisher | Carol Davila University Press |
record_format | MEDLINE/PubMed |
spelling | pubmed-30189632011-03-03 Intracranial hypertension: classification and patterns of evolution Iencean, SM Ciurea, AV J Med Life Review Intracranial hypertension (ICH) was systematized in four categories according to its aetiology and pathogenic mechanisms: parenchymatous ICH with an intrinsic cerebral cause; vascular ICH, which has its aetiology in disorders of cerebral blood circulation; ICH caused by disorders of cerebro–spinal fluid dynamics and idiopathic ICH. The increase of intracranial pressure is the first to happen and then intracranial hypertension develops from this initial effect becoming symptomatic; it then acquires its individuality, surpassing the initial disease. The intracranial hypertension syndrome corresponds to the stage at which the increased intracranial pressure can be compensated and the acute form of intracranial hypertension is equivalent to a decompensated ICH syndrome. The decompensation of intracranial hypertension is a condition of instability and appears when the normal intrinsic ratio of intracranial pressure – time fluctuation is changed. The essential conditions for decompensation of intracranial hypertension are: the speed of intracranial pressure increase over normal values, the highest value of abnormal intracranial pressure and the duration of high ICP values. Medical objectives are preventing ICP from exceeding 20 mm Hg and maintaining a normal cerebral blood flow. The emergency therapy is the same for the acute form but each of the four forms of ICH has a specific therapy, according to the pathogenic mechanism and if possible to aetiology. Carol Davila University Press 2008-04-15 /pmc/articles/PMC3018963/ /pubmed/20108456 Text en ©Carol Davila University Press http://creativecommons.org/licenses/by/2.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use,distribution, and reproduction in any medium, provided the original work is properly cited. |
spellingShingle | Review Iencean, SM Ciurea, AV Intracranial hypertension: classification and patterns of evolution |
title | Intracranial hypertension: classification and patterns of evolution |
title_full | Intracranial hypertension: classification and patterns of evolution |
title_fullStr | Intracranial hypertension: classification and patterns of evolution |
title_full_unstemmed | Intracranial hypertension: classification and patterns of evolution |
title_short | Intracranial hypertension: classification and patterns of evolution |
title_sort | intracranial hypertension: classification and patterns of evolution |
topic | Review |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3018963/ https://www.ncbi.nlm.nih.gov/pubmed/20108456 |
work_keys_str_mv | AT ienceansm intracranialhypertensionclassificationandpatternsofevolution AT ciureaav intracranialhypertensionclassificationandpatternsofevolution |