The kidney in cirrhosis with portal hypertension

Ascites, dilutional hyponatremia and hepatorenal syndrome are three clinical manifestations of the same physiopathological disorder: cirrhotic portal hypertension, hyperproduction of nitric oxide, arterial vasodilation with reduction of efficient arterial volume, which have as consequences renal vas...

Descripción completa

Detalles Bibliográficos
Autores principales: Olteanu, D, Lupu, D
Formato: Texto
Lenguaje:English
Publicado: Carol Davila University Press 2010
Materias:
General Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3019057/
https://www.ncbi.nlm.nih.gov/pubmed/20968204
_version_ 1782196155159937024
author Olteanu, D
Lupu, D
author_facet Olteanu, D
Lupu, D
author_sort Olteanu, D
collection PubMed
description Ascites, dilutional hyponatremia and hepatorenal syndrome are three clinical manifestations of the same physiopathological disorder: cirrhotic portal hypertension, hyperproduction of nitric oxide, arterial vasodilation with reduction of efficient arterial volume, which have as consequences renal vasoconstriction, sympathetic stimulation, the stimulation of renin–angiotensin–aldosteron system and of vasopressin secretion. In dilutional hyponatremia, the selective receptor V2 (vasopressin 1) antagonists may be efficient according to Spanish and American specialists and also according to personal experience [6,9]. Abbreviations:Ly = lymphatic hyperproduction,Alb = hypoalbuminaemia, NO = nitric oxide,Sy = sympathetic (nervous) system,HRS = hepatorenal syndrome
format Text
id pubmed-3019057
institution National Center for Biotechnology Information
language English
publishDate 2010
publisher Carol Davila University Press
record_format MEDLINE/PubMed
spelling pubmed-30190572011-03-03 The kidney in cirrhosis with portal hypertension Olteanu, D Lupu, D J Med Life General Article Ascites, dilutional hyponatremia and hepatorenal syndrome are three clinical manifestations of the same physiopathological disorder: cirrhotic portal hypertension, hyperproduction of nitric oxide, arterial vasodilation with reduction of efficient arterial volume, which have as consequences renal vasoconstriction, sympathetic stimulation, the stimulation of renin–angiotensin–aldosteron system and of vasopressin secretion. In dilutional hyponatremia, the selective receptor V2 (vasopressin 1) antagonists may be efficient according to Spanish and American specialists and also according to personal experience [6,9]. Abbreviations:Ly = lymphatic hyperproduction,Alb = hypoalbuminaemia, NO = nitric oxide,Sy = sympathetic (nervous) system,HRS = hepatorenal syndrome Carol Davila University Press 2010-05-15 2010-05-25 /pmc/articles/PMC3019057/ /pubmed/20968204 Text en ©Carol Davila University Press http://creativecommons.org/licenses/by/2.0/ This is an open-access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle General Article
Olteanu, D
Lupu, D
The kidney in cirrhosis with portal hypertension
title The kidney in cirrhosis with portal hypertension
title_full The kidney in cirrhosis with portal hypertension
title_fullStr The kidney in cirrhosis with portal hypertension
title_full_unstemmed The kidney in cirrhosis with portal hypertension
title_short The kidney in cirrhosis with portal hypertension
title_sort kidney in cirrhosis with portal hypertension
topic General Article
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3019057/
https://www.ncbi.nlm.nih.gov/pubmed/20968204
work_keys_str_mv AT olteanud thekidneyincirrhosiswithportalhypertension
AT lupud thekidneyincirrhosiswithportalhypertension
AT olteanud kidneyincirrhosiswithportalhypertension
AT lupud kidneyincirrhosiswithportalhypertension

Ejemplares similares