Cargando…
Calpain activation through galectin-3 inhibition sensitizes prostate cancer cells to cisplatin treatment
Prostate cancer will develop chemoresistance following a period of chemotherapy. This is due, in part, to the acquisition of antiapoptotic properties by the cancer cells and, therefore, development of novel strategies for treatment is of critical need. Here, we attempt to clarify the role of the ant...
Autores principales: | Wang, Y, Nangia-Makker, P, Balan, V, Hogan, V, Raz, A |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group
2010
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3032324/ https://www.ncbi.nlm.nih.gov/pubmed/21368866 http://dx.doi.org/10.1038/cddis.2010.79 |
Ejemplares similares
-
Galectin-3 regulates p21 stability in human prostate cancer cells
por: Wang, Yi, et al.
Publicado: (2012) -
Chimeric galectin-3 and collagens: Biomarkers and potential therapeutic targets in fibroproliferative diseases
por: Nangia-Makker, Pratima, et al.
Publicado: (2022) -
Galectins as Cancer Biomarkers
por: Balan, Vitaly, et al.
Publicado: (2010) -
Regulation of Tumor Progression by Extracellular Galectin-3
por: Nangia-Makker, Pratima, et al.
Publicado: (2008) -
Galectin-3: a possible complementary marker to the PSA blood test
por: Balan, Vitaly, et al.
Publicado: (2013)