Cargando…
Reactivity against Complementary Proteinase-3 Is Not Increased in Patients with PR3-ANCA-Associated Vasculitis
The etiology of anti-neutrophil cytoplasmic antibodies (ANCA) associated vasculitides (AAV) is unknown, but the association between infections and autoimmunity has been studied extensively. In 2004, a novel theory was proposed that could link infection and autoimmunity. This ‘theory of autoantigen c...
Autores principales: | Tadema, Henko, Kallenberg, Cees G. M., Stegeman, Coen A., Heeringa, Peter |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3060099/ https://www.ncbi.nlm.nih.gov/pubmed/21437233 http://dx.doi.org/10.1371/journal.pone.0017972 |
Ejemplares similares
-
Increased Expression of Toll-Like Receptors by Monocytes and Natural Killer Cells in ANCA-Associated Vasculitis
por: Tadema, Henko, et al.
Publicado: (2011) -
Membrane expression of neutrophil proteinase 3 (PR3) is associated with relapse in PR3-ANCA related vasculitis
por: Rarok, AA, et al.
Publicado: (2002) -
Vasculitis and infections: Contribution to the issue of autoimmunity reviews devoted to “autoimmunity and infection”
por: Kallenberg, Cees G.M., et al.
Publicado: (2008) -
Specific IgG glycosylation differences precede relapse in PR3-ANCA associated vasculitis patients with and without ANCA rise
por: Wojcik, Iwona, et al.
Publicado: (2023) -
Circulating autoreactive proteinase 3(+) B cells and tolerance checkpoints in ANCA-associated vasculitis
por: Berti, Alvise, et al.
Publicado: (2021)