Cargando…
HDAC-mediated Deacetylation of NF-κB is Critical for Schwann cell Myelination
Schwann cell myelination is tightly regulated by timely expression of key transcriptional regulators that respond to specific environmental cues, yet molecular mechanisms underlying such a process are poorly understood. Here, we report that HDAC1/2-regulated acetylation state of NF-κB is critical in...
Autores principales: | Chen, Ying, Wang, Haibo, Yoon, Sung Ok, Xu, Xiaomei, Hottiger, Michael, Svaren, John, Nave, Klaus A., Kim, Haesun A., Olson, Eric N., Lu, Q. Richard |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3074381/ https://www.ncbi.nlm.nih.gov/pubmed/21423191 http://dx.doi.org/10.1038/nn.2780 |
Ejemplares similares
-
Tissue Inhibitor of Metalloproteinase-3 Promotes Schwann Cell Myelination
por: Kim, Jihyun, et al.
Publicado: (2017) -
HDAC3 Regulates the Transition to the Homeostatic Myelinating Schwann Cell State
por: Rosenberg, Laura H., et al.
Publicado: (2018) -
Sodium phenylbutyrate inhibits Schwann cell inflammation via HDAC and NFκB to promote axonal regeneration and remyelination
por: Yadav, Anjali, et al.
Publicado: (2021) -
Zeb2 is essential for Schwann cell differentiation, myelination and
nerve repair
por: Quintes, Susanne, et al.
Publicado: (2016) -
Control of Schwann cell myelination
por: Jessen, Kristján R, et al.
Publicado: (2010)