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The distribution of CTL epitopes in HIV-1 appears to be random, and similar to that of other proteomes
BACKGROUND: HIV-1 viruses are highly capable of mutating their proteins to escape the presentation of CTL epitopes in their current host. Upon transmission to another host, some escape mutations revert, but other remain stable in the virus sequence for at least several years. Depending on the rate o...
| Autores principales: | , , |
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| Formato: | Texto |
| Lenguaje: | English |
| Publicado: |
BioMed Central
2009
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| Materias: | |
| Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3087517/ https://www.ncbi.nlm.nih.gov/pubmed/19653887 http://dx.doi.org/10.1186/1471-2148-9-184 |