Cargando…
CD38 Exacerbates Focal Cytokine Production, Postischemic Inflammation and Brain Injury after Focal Cerebral Ischemia
BACKGROUND: Converging evidence suggests that inflammatory processes significantly influence brain injury and clinical impairment in ischemic stroke. Although early studies suggested a key role of lymphocytes, recent data has emphasized the orchestrating function of innate immunity, i.e., macrophage...
Autores principales: | Choe, Chi-un, Lardong, Kerstin, Gelderblom, Mathias, Ludewig, Peter, Leypoldt, Frank, Koch-Nolte, Friedrich, Gerloff, Christian, Magnus, Tim |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Public Library of Science
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3097994/ https://www.ncbi.nlm.nih.gov/pubmed/21625615 http://dx.doi.org/10.1371/journal.pone.0019046 |
Ejemplares similares
-
Correction: CD38 Exacerbates Focal Cytokine Production, Postischemic Inflammation and Brain Injury after Focal Cerebral Ischemia
por: Choe, Chi-un, et al.
Publicado: (2011) -
Deficiency in Serine Protease Inhibitor Neuroserpin Exacerbates Ischemic Brain Injury by Increased Postischemic Inflammation
por: Gelderblom, Mathias, et al.
Publicado: (2013) -
Depletion of microglia exacerbates postischemic inflammation and brain injury
por: Jin, Wei-Na, et al.
Publicado: (2017) -
Dimethylarginine Dimethylaminohydrolase-1 Transgenic Mice Are Not Protected from Ischemic Stroke
por: Leypoldt, Frank, et al.
Publicado: (2009) -
Berberine confers neuroprotection in coping with focal cerebral ischemia by targeting inflammatory cytokines
por: Maleki, Solmaz Nasseri, et al.
Publicado: (2018)