Cargando…
TRPC channel modulation in podocytes—inching toward novel treatments for glomerular disease
Glomerular kidney disease is a major healthcare burden and considered to represent a sum of disorders that evade a refined and effective treatment. Excellent biological and genetic studies have defined pathways that go awry in podocytes, which are the regulatory cells of the glomerular filter. The q...
Autores principales: | El Hindi, Shafic, Reiser, Jochen |
---|---|
Formato: | Texto |
Lenguaje: | English |
Publicado: |
Springer-Verlag
2010
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3098353/ https://www.ncbi.nlm.nih.gov/pubmed/21161284 http://dx.doi.org/10.1007/s00467-010-1718-4 |
Ejemplares similares
-
Podocyte-Specific Overexpression of Wild Type or Mutant Trpc6 in Mice Is Sufficient to Cause Glomerular Disease
por: Krall, Paola, et al.
Publicado: (2010) -
Cytoskeleton Rearrangements Modulate TRPC6 Channel Activity in Podocytes
por: Shalygin, Alexey, et al.
Publicado: (2021) -
Calcium, TRPC channels, and regulation of the actin cytoskeleton in podocytes: towards a future of targeted therapies
por: Wieder, Nicolas, et al.
Publicado: (2015) -
Podocyte injury in diabetic nephropathy: implications of angiotensin II – dependent activation of TRPC channels
por: Ilatovskaya, Daria V., et al.
Publicado: (2015) -
TRPC5 Channel Inhibition Protects Podocytes in Puromycin-Aminonucleoside Induced Nephrosis Models
por: Zhou, Yiming, et al.
Publicado: (2021)