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Severe Hypocalcemia due to Vitamin D Deficiency after Extended Roux-en-Y Gastric Bypass

Vitamin D deficiency is a well-known comorbidity of obesity that can be exacerbated after bariatric surgery and can predispose the patient for hypocalcemia. Vitamin D and calcium doses to prevent and treat vitamin D deficiency after weight loss surgery are not well defined. We describe a patient who...

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Detalles Bibliográficos
Autores principales: Miñambres, I., Chico, A., Pérez, A.
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2011
Materias:
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3136099/
https://www.ncbi.nlm.nih.gov/pubmed/21772998
http://dx.doi.org/10.1155/2011/141024
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author Miñambres, I.
Chico, A.
Pérez, A.
author_facet Miñambres, I.
Chico, A.
Pérez, A.
author_sort Miñambres, I.
collection PubMed
description Vitamin D deficiency is a well-known comorbidity of obesity that can be exacerbated after bariatric surgery and can predispose the patient for hypocalcemia. Vitamin D and calcium doses to prevent and treat vitamin D deficiency after weight loss surgery are not well defined. We describe a patient who developed severe hypocalcemia due to vitamin D deficiency 5 years after an extended Roux-en-Y gastric bypass for a type II obesity. No precipitating factors were present and malabsorption induced by the bypass was considered to be the main causative factor. High doses of vitamin D and calcium were needed to reach and maintain normal calcium and vitamin D concentrations. This case emphasises the importance of routine screening for vitamin D deficiency in obese individuals and reflects that while consensus does not exist regarding optimal dosage, vitamin D replacement should be tittered based on calcidiol levels.
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spelling pubmed-31360992011-07-19 Severe Hypocalcemia due to Vitamin D Deficiency after Extended Roux-en-Y Gastric Bypass Miñambres, I. Chico, A. Pérez, A. J Obes Case Report Vitamin D deficiency is a well-known comorbidity of obesity that can be exacerbated after bariatric surgery and can predispose the patient for hypocalcemia. Vitamin D and calcium doses to prevent and treat vitamin D deficiency after weight loss surgery are not well defined. We describe a patient who developed severe hypocalcemia due to vitamin D deficiency 5 years after an extended Roux-en-Y gastric bypass for a type II obesity. No precipitating factors were present and malabsorption induced by the bypass was considered to be the main causative factor. High doses of vitamin D and calcium were needed to reach and maintain normal calcium and vitamin D concentrations. This case emphasises the importance of routine screening for vitamin D deficiency in obese individuals and reflects that while consensus does not exist regarding optimal dosage, vitamin D replacement should be tittered based on calcidiol levels. Hindawi Publishing Corporation 2011 2011-05-18 /pmc/articles/PMC3136099/ /pubmed/21772998 http://dx.doi.org/10.1155/2011/141024 Text en Copyright © 2011 I. Miñambres et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
spellingShingle Case Report
Miñambres, I.
Chico, A.
Pérez, A.
Severe Hypocalcemia due to Vitamin D Deficiency after Extended Roux-en-Y Gastric Bypass
title Severe Hypocalcemia due to Vitamin D Deficiency after Extended Roux-en-Y Gastric Bypass
title_full Severe Hypocalcemia due to Vitamin D Deficiency after Extended Roux-en-Y Gastric Bypass
title_fullStr Severe Hypocalcemia due to Vitamin D Deficiency after Extended Roux-en-Y Gastric Bypass
title_full_unstemmed Severe Hypocalcemia due to Vitamin D Deficiency after Extended Roux-en-Y Gastric Bypass
title_short Severe Hypocalcemia due to Vitamin D Deficiency after Extended Roux-en-Y Gastric Bypass
title_sort severe hypocalcemia due to vitamin d deficiency after extended roux-en-y gastric bypass
topic Case Report
url https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3136099/
https://www.ncbi.nlm.nih.gov/pubmed/21772998
http://dx.doi.org/10.1155/2011/141024
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