Cargando…
Accumulation of Kv7.2 channels in putative ectopic transduction zones of mice nerve-end neuromas
BACKGROUND: Modulation of M-type currents has been proposed as a new strategy for the treatment of neuropathic pain due to their role in regulating neuronal excitability. Using electrophysiological techniques we showed previously that the opening of Kv7 channels with retigabine, blocked ectopic disc...
Autores principales: | Roza, Carolina, Castillejo, Sol, Lopez-García, Jose A |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3163195/ https://www.ncbi.nlm.nih.gov/pubmed/21838927 http://dx.doi.org/10.1186/1744-8069-7-58 |
Ejemplares similares
-
Tamoxifen Inhibition of Kv7.2/Kv7.3 Channels
por: Ferrer, Tania, et al.
Publicado: (2013) -
Transcriptional repression of the M channel subunit Kv7.2 in chronic nerve injury
por: Rose, Kirstin, et al.
Publicado: (2011) -
Axonal Kv7.2/7.3 channels: Caught in the act
por: Kole, Maarten HP, et al.
Publicado: (2014) -
Calmodulin: A Multitasking Protein in Kv7.2 Potassium Channel Functions
por: Alaimo, Alessandro, et al.
Publicado: (2018) -
Ethanol inhibits Kv7.2/7.3 channel open probability by reducing the PI(4,5)P(2) sensitivity of Kv7.2 subunit
por: Kim, Kwon-Woo, et al.
Publicado: (2021)