Cargando…
Fiber Tracts Anomalies in APPxPS1 Transgenic Mice Modeling Alzheimer's Disease
Amyloid beta (Aβ) peptides are known to accumulate in the brain of patients with Alzheimer's disease (AD). However, the link between brain amyloidosis and clinical symptoms has not been elucidated and could be mediated by secondary neuropathological alterations such as fiber tracts anomalies. I...
Autores principales: | Chen, H., Epelbaum, S., Delatour, B. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
SAGE-Hindawi Access to Research
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3170810/ https://www.ncbi.nlm.nih.gov/pubmed/21912744 http://dx.doi.org/10.4061/2011/281274 |
Ejemplares similares
-
Activity-induced MEMRI cannot detect functional brain anomalies in the APPxPS1-Ki mouse model of Alzheimer’s disease
por: Androuin, Alexandre, et al.
Publicado: (2019) -
Electric polarisability of hydrogen atom with Z.ge appx.137
por: Golbraih, E I, et al.
Publicado: (1983) -
In Vivo Imaging Biomarkers in Mouse Models of Alzheimer's Disease: Are We Lost in Translation or Breaking Through?
por: Delatour, Benoît, et al.
Publicado: (2010) -
Vaccine Development to Treat Alzheimer's Disease Neuropathology in APP/PS1 Transgenic Mice
por: Carrera, Iván, et al.
Publicado: (2012) -
Untargeted lipidomics reveals progression of early Alzheimer’s disease in APP/PS1 transgenic mice
por: Zhang, Xueju, et al.
Publicado: (2020)