Cargando…
Cell death regulation during influenza A virus infection by matrix (M1) protein: a model of viral control over the cellular survival pathway
During early infection, viruses activate cellular stress-response proteins such as heat-shock proteins (Hsps) to counteract apoptosis, but later on, they modulate these proteins to stimulate apoptosis for efficient viral dissemination. Hsp70 has been attributed to modulate viral entry, transcription...
Autores principales: | Halder, U C, Bagchi, P, Chattopadhyay, S, Dutta, D, Chawla-Sarkar, M |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Nature Publishing Group
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3186897/ https://www.ncbi.nlm.nih.gov/pubmed/21881599 http://dx.doi.org/10.1038/cddis.2011.75 |
Ejemplares similares
-
Matrix and Backstage: Cellular Substrates for Viral Vaccines
por: Jordan, Ingo, et al.
Publicado: (2014) -
Quercetin, a flavonoid, combats rotavirus infection by deactivating rotavirus-induced pro-survival NF-κB pathway
por: Banerjee, Shreya, et al.
Publicado: (2022) -
Differential modulation of cellular death and survival pathways by conjugated bile acids
por: Torchia, Enrique C, et al.
Publicado: (2001) -
Inhibition of Influenza A Virus Infection by Fucoidan Targeting Viral Neuraminidase and Cellular EGFR Pathway
por: Wang, Wei, et al.
Publicado: (2017) -
Influenza viral matrix 1 protein aggravates viral pathogenicity by inducing TLR4-mediated reactive oxygen species production and apoptotic cell death
por: Kim, Chang-Ung, et al.
Publicado: (2023)