Cargando…

Low levels of the nerve growth factor receptor TrkA in JIA: a possible defect in a novel anti-inflammatory mechanism

Mostrar otras versiones (1)

Detalles Bibliográficos
Autores principales:	Minnone, G, Strippoli, R, Prencipe, G, De Pasquale, L, Bracaglia, C, Insalaco, A, De Benedetti, F, Bracci-Laudiero, L
Formato:	Online Artículo Texto
Lenguaje:	English
Publicado:	BioMed Central 2011
Materias:	Oral Presentation
Acceso en línea:	https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3194445/ http://dx.doi.org/10.1186/1546-0096-9-S1-O9

Ejemplares similares

Low levels of the nerve growth factor receptor TrkA in JIA: a possible defect in a novel anti-inflammatory mechanism
por: Minnone, G, et al.
Publicado: (2011)

P01-050 – Anakinra in systemic JIA: single center experience
por: Pardeo, M, et al.
Publicado: (2013)

TrkA Co-Receptors: The Janus Face of TrkA?
por: Trouvilliez, Sarah, et al.
Publicado: (2023)

NGF and Its Receptors in the Regulation of Inflammatory Response
por: Minnone, Gaetana, et al.
Publicado: (2017)

S100A12 as diagnostic tool in the differential diagnosis of sJIA associated MAS vs. hereditary or acquired HLH
por: Holzinger, D, et al.
Publicado: (2015)

Effects of pre- and postnatal deletion of the transcription factor NKx2-1 on the expression of NGF, trkA, trkB and p75NTR in mice
por: Ersözlü, S, et al.
Publicado: (2015)

PReS-FINAL-2183: IL-6 amplifies toll like receptor mediated cytokine and chemokine production: implications for the pathogenesis of rheumatic inflammatory diseases
por: Caiello, I, et al.
Publicado: (2013)

Deregulation of IL-1β axis in peripheral blood mononuclear cells from patients with Chronic Recurrent Multifocal Osteomyelitis
por: Scianaro, Roberta, et al.
Publicado: (2011)

Interferon-gamma (IFNy) in macrophage activation syndrome (MAS) associated with systemic juvenile idiopathic arthritis (sJIA). High levels in patients and a role in a murine mas model
por: Bracaglia, Claudia, et al.
Publicado: (2014)

Neutralization of Interferon-gamma is efficacious in a mouse model of HLH secondary to chronic inflammation
por: Prencipe, G, et al.
Publicado: (2015)

Deregulation of the IL-1β axis in chronic recurrent multifocal osteomyelitis
por: Scianaro, Roberta, et al.
Publicado: (2014)

NGF – the TrkA to successful pain treatment
por: Kumar, Vinayak, et al.
Publicado: (2012)

OR6-005 – Cystine crystals activate inflammasomes
por: Prencipe, G, et al.
Publicado: (2013)

ProNGF-p75NTR axis plays a proinflammatory role in inflamed joints: a novel pathogenic mechanism in chronic arthritis
por: Minnone, Gaetana, et al.
Publicado: (2017)

PReS-FINAL-2096: Herpes virus infections in patients with juvenile idiopathic arthritis (JIA) treated with etanercept
por: Nicolai, R, et al.
Publicado: (2013)

Pincher, a pinocytic chaperone for nerve growth factor/TrkA signaling endosomes
por: Shao, Yufang, et al.
Publicado: (2002)

Functional significance of nerve growth factor and its receptor (TrkA) in inflammatory arthritis
por: Raychaudhuri, Smriti K, et al.
Publicado: (2010)

Infliximab to treat chronic uveitis in juvenile idiopathic arthritis (JIA)
por: Bracaglia, C, et al.
Publicado: (2008)

Inflammatory Cytokine response in a cohort of patients carrying novel NLRP12 variants
por: Raganelli, L, et al.
Publicado: (2015)

Combinatorial Assembly of Small Molecules into Bivalent Antagonists of TrkC or TrkA Receptors
por: Brahimi, Fouad, et al.
Publicado: (2014)

TrkA regulates the regenerative capacity of bone marrow stromal stem cells in nerve grafts
por: Zheng, Mei-Ge, et al.
Publicado: (2019)

Expression of Nerve Growth Factor and Its Receptor TrkA in the Reproductive System of Adult Zebrafish
por: Cacialli, Pietro
Publicado: (2022)

TrkA inhibitor promotes motor functional regeneration of recurrent laryngeal nerve by suppression of sensory nerve regeneration
por: Suzuki, Hiroshi, et al.
Publicado: (2020)

NGF FLIPs TrkA onto the death TRAIL in neuroblastoma cells
por: Ruggeri, P, et al.
Publicado: (2016)

PReS-FINAL-2142: Predictors of persistent remission following etanercept (ETN) withdrawal in patients with juvenile idiopathic arthritis (JIA)
por: Perfetti, F, et al.
Publicado: (2013)

NGF Causes TrkA to Specifically Attract Microtubules to Lipid Rafts
por: Pryor, Shona, et al.
Publicado: (2012)

Transcytosis of TrkA leads to diversification of dendritic signaling endosomes
por: Barford, Kelly, et al.
Publicado: (2018)

Pronerve Growth Factor Induces Angiogenesis via Activation of TrkA: Possible Role in Proliferative Diabetic Retinopathy
por: Elshaer, Sally L., et al.
Publicado: (2013)

A neuroprotective agent that inactivates prodegenerative TrkA and preserves mitochondria
por: Feinberg, Konstantin, et al.
Publicado: (2017)

Low molecular weight, non-peptidic agonists of TrkA receptor with NGF-mimetic activity
por: Scarpi, D, et al.
Publicado: (2012)

Low molecular weight, non-peptidic agonists of TrkA receptor with NGF-mimetic activity
por: Scarpi, D, et al.
Publicado: (2012)

NGF-stimulated retrograde transport of trkA in the mammalian nervous system
Publicado: (1995)

Sympathetic neurons secrete retrogradely transported TrkA on extracellular vesicles
por: Mason, Ashley J., et al.
Publicado: (2023)

Enhanced TrkA signaling impairs basal forebrain-dependent behavior
por: Calvo-Enrique, Laura, et al.
Publicado: (2023)

TrkA(+) Neurons Induce Pathologic Regeneration After Soft Tissue Trauma
por: Cherief, Masnsen, et al.
Publicado: (2022)

Sensory Neuron-Specific Deletion of Tropomyosin Receptor Kinase A (TrkA) in Mice Abolishes Osteoarthritis (OA) Pain via NGF/TrkA Intervention of Peripheral Sensitization
por: O-Sullivan, InSug, et al.
Publicado: (2022)

Expression of Nerve Growth Factor (NGF), TrkA, and p75(NTR) in Developing Human Fetal Teeth
por: Mitsiadis, Thimios A., et al.
Publicado: (2016)

Dynamic expression of nerve growth factor and its receptor TrkA after subarachnoid hemorrhage in rat brain
por: Song, Jin-ning, et al.
Publicado: (2016)

Nerve growth factor and its receptor tyrosine kinase TrkA are overexpressed in cervical squamous cell carcinoma
por: Faulkner, Sam, et al.
Publicado: (2020)

Engineered Sensory Nerve Guides Self‐Adaptive Bone Healing via NGF‐TrkA Signaling Pathway
por: Zhang, Zengjie, et al.
Publicado: (2023)

Cannot write session to /tmp/vufind_sessions/sess_388eln7j85ilt94edqtprcueef