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Pathophysiology of intensive care unit-acquired anemia
The formation of red blood cells (RBCs) in the bone marrow is regulated by erythropoietin in response to a cascade of events. Anemia in the intensive care unit can be caused by a host of factors. Patients in the intensive care unit may have decreased RBC production and a blunted response to erythrop...
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Formato: | Online Artículo Texto |
Lenguaje: | English |
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BioMed Central
2004
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Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3226145/ https://www.ncbi.nlm.nih.gov/pubmed/15196314 http://dx.doi.org/10.1186/cc2410 |
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author | Fink, Mitchell P |
author_facet | Fink, Mitchell P |
author_sort | Fink, Mitchell P |
collection | PubMed |
description | The formation of red blood cells (RBCs) in the bone marrow is regulated by erythropoietin in response to a cascade of events. Anemia in the intensive care unit can be caused by a host of factors. Patients in the intensive care unit may have decreased RBC production and a blunted response to erythropoietin. Administration of recombinant human erythropoietin may stimulate erythropoiesis, increase hematocrit levels and hemoglobin concentration, and reduce the need for RBC transfusions. |
format | Online Article Text |
id | pubmed-3226145 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2004 |
publisher | BioMed Central |
record_format | MEDLINE/PubMed |
spelling | pubmed-32261452011-11-30 Pathophysiology of intensive care unit-acquired anemia Fink, Mitchell P Crit Care Review The formation of red blood cells (RBCs) in the bone marrow is regulated by erythropoietin in response to a cascade of events. Anemia in the intensive care unit can be caused by a host of factors. Patients in the intensive care unit may have decreased RBC production and a blunted response to erythropoietin. Administration of recombinant human erythropoietin may stimulate erythropoiesis, increase hematocrit levels and hemoglobin concentration, and reduce the need for RBC transfusions. BioMed Central 2004 2004-06-14 /pmc/articles/PMC3226145/ /pubmed/15196314 http://dx.doi.org/10.1186/cc2410 Text en Copyright ©2004 BioMed Central Ltd |
spellingShingle | Review Fink, Mitchell P Pathophysiology of intensive care unit-acquired anemia |
title | Pathophysiology of intensive care unit-acquired anemia |
title_full | Pathophysiology of intensive care unit-acquired anemia |
title_fullStr | Pathophysiology of intensive care unit-acquired anemia |
title_full_unstemmed | Pathophysiology of intensive care unit-acquired anemia |
title_short | Pathophysiology of intensive care unit-acquired anemia |
title_sort | pathophysiology of intensive care unit-acquired anemia |
topic | Review |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3226145/ https://www.ncbi.nlm.nih.gov/pubmed/15196314 http://dx.doi.org/10.1186/cc2410 |
work_keys_str_mv | AT finkmitchellp pathophysiologyofintensivecareunitacquiredanemia |