Cargando…
Transition to absence seizures and the role of GABA(A) receptors
Absence seizures appear to be initiated in a putative cortical ‘initiation site’ by the expression of medium-amplitude 5–9 Hz oscillations, which may in part be due to a decreased phasic GABA(A) receptor function. These oscillations rapidly spread to other cortical areas and to the thalamus, leading...
Autores principales: | Crunelli, Vincenzo, Cope, David W., Terry, John R. |
---|---|
Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
Elsevier Science Publishers
2011
|
Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3227737/ https://www.ncbi.nlm.nih.gov/pubmed/21889315 http://dx.doi.org/10.1016/j.eplepsyres.2011.07.011 |
Ejemplares similares
-
Augmentation of Tonic GABA(A) Inhibition in Absence Epilepsy: Therapeutic Value of Inverse Agonists at Extrasynaptic GABA(A) Receptors
por: Errington, Adam C., et al.
Publicado: (2011) -
Developmental changes of GABA immunoreactivity in cortico-thalamic networks of an absence seizure model
por: Bombardi, Cristiano, et al.
Publicado: (2018) -
Enhanced tonic GABA(A) inhibition in typical absence epilepsy
por: Cope, David W., et al.
Publicado: (2009) -
Aberrant GABA(A) Receptor-Mediated Inhibition in Cortico-Thalamic Networks of Succinic Semialdehyde Dehydrogenase Deficient Mice
por: Errington, Adam C., et al.
Publicado: (2011) -
Genetic rescue of absence seizures
por: McCafferty, Cian, et al.
Publicado: (2018)