Preeclampsia, Hypoxia, Thrombosis, and Inflammation

Reductions in uteroplacental flow initiate a cascade of molecular effects leading to hypoxia, thrombosis, inflammation, and endothelial cell dysfunction resulting in untoward pregnancy outcomes. In this review, we detail these effects and their relationship to preeclampsia (PE) and intrauterine grow...

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Detalles Bibliográficos
Autores principales: Shamshirsaz, Amir A., Paidas, Michael, Krikun, Graciela
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Hindawi Publishing Corporation 2012
Materias:
Review Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3235807/
https://www.ncbi.nlm.nih.gov/pubmed/22175023
http://dx.doi.org/10.1155/2012/374047
Descripción
Sumario:Reductions in uteroplacental flow initiate a cascade of molecular effects leading to hypoxia, thrombosis, inflammation, and endothelial cell dysfunction resulting in untoward pregnancy outcomes. In this review, we detail these effects and their relationship to preeclampsia (PE) and intrauterine growth restriction (IUGR).

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