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Neuroinflammation in Alzheimer's disease wanes with age
BACKGROUND: Inflammation is a prominent feature in Alzheimer's disease (AD). It has been proposed that aging has an effect on the function of inflammation in the brain, thereby contributing to the development of age-related diseases like AD. However, the age-dependent relationship between infla...
Autores principales: | , , , , |
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Formato: | Online Artículo Texto |
Lenguaje: | English |
Publicado: |
BioMed Central
2011
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Materias: | |
Acceso en línea: | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3248382/ https://www.ncbi.nlm.nih.gov/pubmed/22152162 http://dx.doi.org/10.1186/1742-2094-8-171 |
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author | Hoozemans, Jeroen JM Rozemuller, Annemieke JM van Haastert, Elise S Eikelenboom, Piet van Gool, Willem A |
author_facet | Hoozemans, Jeroen JM Rozemuller, Annemieke JM van Haastert, Elise S Eikelenboom, Piet van Gool, Willem A |
author_sort | Hoozemans, Jeroen JM |
collection | PubMed |
description | BACKGROUND: Inflammation is a prominent feature in Alzheimer's disease (AD). It has been proposed that aging has an effect on the function of inflammation in the brain, thereby contributing to the development of age-related diseases like AD. However, the age-dependent relationship between inflammation and clinical phenotype of AD has never been investigated. METHODS: In this study we have analysed features of the neuroinflammatory response in clinically and pathologically confirmed AD and control cases in relation to age (range 52-97 years). The mid-temporal cortex of 19 controls and 19 AD cases was assessed for the occurrence of microglia and astrocytes by immunohistochemistry using antibodies directed against CD68 (KP1), HLA class II (CR3/43) and glial fibrillary acidic protein (GFAP). RESULTS: By measuring the area density of immunoreactivity we found significantly more microglia and astrocytes in AD cases younger than 80 years compared to older AD patients. In addition, the presence of KP1, CR3/43 and GFAP decreases significantly with increasing age in AD. CONCLUSION: Our data suggest that the association between neuroinflammation and AD is stronger in relatively young patients than in the oldest patients. This age-dependent relationship between inflammation and clinical phenotype of AD has implications for the interpretation of biomarkers and treatment of the disease. |
format | Online Article Text |
id | pubmed-3248382 |
institution | National Center for Biotechnology Information |
language | English |
publishDate | 2011 |
publisher | BioMed Central |
record_format | MEDLINE/PubMed |
spelling | pubmed-32483822011-12-30 Neuroinflammation in Alzheimer's disease wanes with age Hoozemans, Jeroen JM Rozemuller, Annemieke JM van Haastert, Elise S Eikelenboom, Piet van Gool, Willem A J Neuroinflammation Research BACKGROUND: Inflammation is a prominent feature in Alzheimer's disease (AD). It has been proposed that aging has an effect on the function of inflammation in the brain, thereby contributing to the development of age-related diseases like AD. However, the age-dependent relationship between inflammation and clinical phenotype of AD has never been investigated. METHODS: In this study we have analysed features of the neuroinflammatory response in clinically and pathologically confirmed AD and control cases in relation to age (range 52-97 years). The mid-temporal cortex of 19 controls and 19 AD cases was assessed for the occurrence of microglia and astrocytes by immunohistochemistry using antibodies directed against CD68 (KP1), HLA class II (CR3/43) and glial fibrillary acidic protein (GFAP). RESULTS: By measuring the area density of immunoreactivity we found significantly more microglia and astrocytes in AD cases younger than 80 years compared to older AD patients. In addition, the presence of KP1, CR3/43 and GFAP decreases significantly with increasing age in AD. CONCLUSION: Our data suggest that the association between neuroinflammation and AD is stronger in relatively young patients than in the oldest patients. This age-dependent relationship between inflammation and clinical phenotype of AD has implications for the interpretation of biomarkers and treatment of the disease. BioMed Central 2011-12-07 /pmc/articles/PMC3248382/ /pubmed/22152162 http://dx.doi.org/10.1186/1742-2094-8-171 Text en Copyright ©2011 Hoozemans et al; licensee BioMed Central Ltd. http://creativecommons.org/licenses/by/2.0 This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. |
spellingShingle | Research Hoozemans, Jeroen JM Rozemuller, Annemieke JM van Haastert, Elise S Eikelenboom, Piet van Gool, Willem A Neuroinflammation in Alzheimer's disease wanes with age |
title | Neuroinflammation in Alzheimer's disease wanes with age |
title_full | Neuroinflammation in Alzheimer's disease wanes with age |
title_fullStr | Neuroinflammation in Alzheimer's disease wanes with age |
title_full_unstemmed | Neuroinflammation in Alzheimer's disease wanes with age |
title_short | Neuroinflammation in Alzheimer's disease wanes with age |
title_sort | neuroinflammation in alzheimer's disease wanes with age |
topic | Research |
url | https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3248382/ https://www.ncbi.nlm.nih.gov/pubmed/22152162 http://dx.doi.org/10.1186/1742-2094-8-171 |
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