Prevention of neonatal oxygen-induced brain damage by reduction of intrinsic apoptosis

Within the last decade, it became clear that oxygen contributes to the pathogenesis of neonatal brain damage, leading to neurocognitive impairment of prematurely born infants in later life. Recently, we have identified a critical role for receptor-mediated neuronal apoptosis in the immature rodent b...

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Detalles Bibliográficos
Autores principales: Sifringer, M, Bendix, I, Börner, C, Endesfelder, S, von Haefen, C, Kalb, A, Holifanjaniaina, S, Prager, S, Schlager, G W, Keller, M, Jacotot, E, Felderhoff-Mueser, U
Formato: Online Artículo Texto
Lenguaje:English
Publicado: Nature Publishing Group 2012
Materias:
Original Article
Acceso en línea:https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3270267/
https://www.ncbi.nlm.nih.gov/pubmed/22237207
http://dx.doi.org/10.1038/cddis.2011.133

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